Journal of ecological engineering

Могу journal of ecological engineering верно!

However, this proportion increased to 0. The short-term use of mupirocin has a strongly reductive effect on staphylococcal infection in chronic rhinosinusitis. Citation: Kim JS, Kwon SH (2016) Mupirocin in the Treatment of Staphylococcal Infections in Chronic Rhinosinusitis: A Meta-Analysis. PLoS ONE 11(12): e0167369. Funding: This paper was supported by a fund of the Biomedical Research Institute at Chonbuk National University Hospital.

However, classic saline irrigation and oral antibiotics have a limited effect on these refractory cases. Of these agents, mupirocin also human genome significant anti-staphylococcal activity.

In this study, our purpose was to evaluate the efficacy of saline irrigation with mupirocin to treat recalcitrant CRS using a systematic review and meta-analysis. This is a systematic retrospective review of previously published articles, and no patient identifiable details are included.

Institutional review board approval and patient consent were not required due to the nature of this study. The MEDLINE, EMBASE and Cochrane databases journal of ecological engineering searched for eligible studies published up to and including December 2015. Studies were excluded if: (1) the treatment modalities contained other topical agents; (2) the article was not written journal of ecological engineering English; (3) the study had no relation to sinusitis; (4) the study included in vitro studies; journal of ecological engineering the study had duplicate data or incomplete data journal of ecological engineering calculating the effect sizes; (6) the study was an unpublished trial.

Two authors independently extracted information from all eligible studies. Any disparities were resolved by consensus. The proportion of treatment failure cases in the experimental group was obtained by dividing the number of cases with treatment failure by the total number of cases in the study.

The proportion of treatment failure cases in the control group was calculated using the same method. The effect size was represented by the risk ratio of residual staphylococcal infection, which was compared between the mupirocin group and the control group.

The standard error was also calculated for each clinical outcome measure. The random effects model was used considering the effects from different locations, populations, and heterogenous research groups, which were the main causes of the within-study and between-study variations. Heterogeneity between studies was assessed using the I2 statistic. Potential publication bias was investigated using funnel plots. A sensitivity analysis was carried out to identify any outlier studies.

The literature search identified 215 articles. The PRISMA flow diagram of this systematic review is shown in Fig 1. Twelve duplicated records were also excluded. The remaining journal of ecological engineering articles qualified for full-text reading, and these were systematically reviewed. After reviewing the journal of ecological engineering text, 24 publications were excluded because they failed to meet our eligibility criteria (eight articles did not include mupirocin journal of ecological engineering, nine had insufficient data, six had abstractive narration, and one was a poster presentation).

Therefore, six articles were finally included in our qualitative analysis (Table 1). Of these six studies, three studies had no control group.

Therefore, three articles were used for effect comparison. The pooled risk difference was calculated to be -0. In the overall comparison, the pooled risk ratio and the stratified analyses were not significantly changed, indicating a gilead sciences limited and robust outcome (Fig 4A).

The pooled risk ratio in the overall comparison was not significantly changed, indicating a stable outcome. The proportion of residual Staphylococcus aureus was 0. After the first month, the proportion of residual staphylococcal infection was 0. The proportion increased to 0. There are two main theories for the development of recurrent CRS: biofilm formation and superantigen formation.

The pathophysiology of biofilm development in CRS includes both bacterial and host factors.



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